Inducible modulation is usually required for exact investigations and manipulations of dynamic organic processes. Transcription activator-like effectors (TALEs) present a
Transcription activator-like effectors (TALEs) are proteins discovered within the genus Xanthomonas of phytopathogenic micro organism. These proteins enter the nucleus
Non-TAL Effectors From Xanthomonas oryzae pv. oryzae Suppress Peptidoglycan-Triggered MAPK Activation in Rice.
Xanthomonas oryzae pv. oryzae, the causal pathogen of bacterial blight of rice, depends upon its sort III secretion system and related effector
Transcription activator-like effectors (TALEs) include modular programmable DNA binding domains. Fusing TALEs with effector domains creates artificial transcription components (TALE-TFs)
Structural insights into the specific recognition of 5-methylcytosine and 5-hydroxymethylcytosine by TAL effectors.
Transcription activator-like effectors (TALEs) acknowledge DNA by means of repeat-variable diresidues (RVDs) and TALE-DNA interactions are delicate to DNA modifications.
SWEET genes encode sugar transporter proteins and usually perform as susceptibility (S) genes. Consequently, the recessive alleles of those SWEET
DNA transposon-based vectors have emerged as new potential delivery tools in therapeutic gene transfer. Such vectors are now showing promise in
DNA-dependent protein kinase (DNA-PK), which is concerned in DNA double-strand break restore and V(D)J recombination, is comprised of a DNA-targeting element termed Ku
The DNA damage response kinases DNA-dependent protein kinase (DNA-PK) and ataxia telangiectasia mutated (ATM) Are stimulated by bulky adduct-containing DNA.
A wide range of environmental, carcinogenic, and chemotherapeutic brokers kind bulky lesions on DNA that activate DNA damage checkpoint signaling pathways in human cells.
Loss of epithelial differentiation and gain of invasiveness correlates with tyrosine phosphorylation of the E-cadherin/beta-catenin complex in cells transformed with a temperature-sensitive v-SRC gene.
Loss of histotypic group of epithelial cells is a frequent characteristic in regular growth in addition to in the invasion