Inducible modulation is usually required for exact investigations and manipulations of dynamic organic processes. Transcription activator-like effectors (TALEs) present a
Transcription activator-like effectors (TALEs) are proteins discovered within the genus Xanthomonas of phytopathogenic micro organism. These proteins enter the nucleus
Non-TAL Effectors From Xanthomonas oryzae pv. oryzae Suppress Peptidoglycan-Triggered MAPK Activation in Rice.
Xanthomonas oryzae pv. oryzae, the causal pathogen of bacterial blight of rice, depends upon its sort III secretion system and related effector
Transcription activator-like effectors (TALEs) include modular programmable DNA binding domains. Fusing TALEs with effector domains creates artificial transcription components (TALE-TFs)
Structural insights into the specific recognition of 5-methylcytosine and 5-hydroxymethylcytosine by TAL effectors.
Transcription activator-like effectors (TALEs) acknowledge DNA by means of repeat-variable diresidues (RVDs) and TALE-DNA interactions are delicate to DNA modifications.
SWEET genes encode sugar transporter proteins and usually perform as susceptibility (S) genes. Consequently, the recessive alleles of those SWEET
DNA transposon-based vectors have emerged as new potential delivery tools in therapeutic gene transfer. Such vectors are now showing promise in
DNA-dependent protein kinase (DNA-PK), which is concerned in DNA double-strand break restore and V(D)J recombination, is comprised of a DNA-targeting element termed Ku
The DNA damage response kinases DNA-dependent protein kinase (DNA-PK) and ataxia telangiectasia mutated (ATM) Are stimulated by bulky adduct-containing DNA.
A wide range of environmental, carcinogenic, and chemotherapeutic brokers kind bulky lesions on DNA that activate DNA damage checkpoint signaling pathways in human cells.